Melatonin activates ABCA1 via the BiP/NRF1 pathway to suppress high-cholesterol-induced apoptosis of mesenchymal stem cells

BNIP3L/NIX-mediated mitophagy protects against glucocorticoid-induced synapse defects

Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis

Ethanol-activated CaMKII signaling induces neuronal apoptosis through Drp1-mediated excessive mitochondrial fission and JNK1-dependent NLRP3 inflammasome activation

High glucose-mediated PICALM and mTORC1 modulate processing of amyloid precursor protein via endosomal abnormalities

Sodium butyrate inhibits high cholesterol-induced neuronal amyloidogenesis by modulating NRF2 stabilization-mediated ROS levels: involvement of NOX2 and SOD1

Role of Microtubule-Associated Factors in HIF1 alpha Nuclear Translocation

Melatonin restores Muc2 depletion induced by V. vulnificus VvpM via melatonin receptor 2 coupling with G alpha q

BICD1 mediates HIF1 alpha nuclear translocation in mesenchymal stem cells during hypoxia adaptation

O-cyclic phytosphingosine-1-phosphate stimulates HIF1 alpha-dependent glycolytic reprogramming to enhance the therapeutic potential of mesenchymal stem cells