발행물
컨퍼런스
신경 면역 네트워크 심포지움
2005
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Neurotoxicity of C-terminal fragments of Amyloid precursor protein
Annual Meeting of Society for Neuroscience
2004
Dehydroevodiamine.HCl extracted from Evodia Rutaecarpa Bentham prevents neuronal loss and cognitive dysfunction through antagonizing oxidative stress and excitotoxicities
Annual meeting of Society for Neuroscience
The intracellular domain of amyloid precursor like protein 2 translocalizes into the nucleus with Fe65 and exerts cytotoxicity by inducing glycogen synthase kinase 3beta expression.
Asian PAcific Society for Neurochemistry
C-terminal fragments of amyloid precursor protein exert neurotoxicity by inducing glycogen synthase kinase 3beta expression.