The role of presenilins in the deveopment

Roles of Abeta and carboxyl terminal peptide fragments of APP in Alzheimer's disease

C-terminal fragment of Alzheimer's APP destabilize calcium homeostasis and renders neuronal cells vulnerable to excitotoxicity

APP carboxyl-terminal fragment without or with abeta domain equally induces cytotoxicity in differentiated PC 12 cells and cortical neurons.

Silica increases cytosolic calcium and causes injury in renal cell lines

Inhibition of the NGF and IL-beta induced-expression of Alzheimer's APP by antisense oligonucleotide

C-terminal fragment od Alzheimer's APP inhibits sodium/calcium exchanger activity in SK-N Sh cells.

C-terminal fragment of APP inhibits calcium uptake into rat brain microsomes by Mg+2-Ca+2 ATPase

Post-ischemic changes in the expression of Alzheimer's APP isoforms in rat cerebral cortex/

Intranephron distribution of cysteine S-conjugate beta lyase activity and its implications for hexachloro-1,3-butadiene-induced nephrotoxicity in rats