,

An Extra-Nuclear Role of Ref-1 in the Regulation of Nitric Oxide Production and Apoptosis in RAW264.7 Cells

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Chloroquine-induced inhibition of NO production is Accomplished by decrease in early secretion of TNF-α without affecting total TNF-α secretion in stimulated macrophages

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NO and TNF-αsecretion in LPS-stimulated murine macrophages are inhibited by the stress response

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HO-1 upregulation represses apoptotic activity by etoposide in human retinal pigment epithelial cells

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Diphenyleneiodonium, a flavoenzyme inhibitor, induces ROS-independent p53 expression and apoptosis in human RPE cells