,

Ovarian cancer growth was induced by 2,3,7,8-tetrachlorodibenzo-ρ-dioxin through the regulation of CYP1A1 gene in an estrogen receptor-dependent pathway in BG-1 ovarian cancer cells

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Progesterone is a potent substance which inhibits the migration of ovarian cancer cells by reducing epithelial-mesenchymal transition via progesterone receptor-dependent pathway

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Bisphenol A-induced epithelial-mesenchymal transition was reversed with a phytoestrogen, genistein via estrogen receptor and downregulation of transforming growth factor-beta signaling pathway

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Epithelial-mesenchymal transition was altered by progesterone in the inhibition of ovarian cancer growth via progesterone receptor involved pathways in an ovarian cancer model

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Cell cycle- and metastasis-related genes were altered by pesticides, fenhexamid and cyprodinil, in ovarian cancer models in an estrogen receptor-dependent manner