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Mycobacterial Tuberculin PPD Antigen Plays an Essential Role in the Activation of ROS-induced p38 MAPK Pathway and Proinflammatory Responses via Toll-

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The Ginsenoside Metabolite Compound K inhibits toll-like receptor 4-mediated inflammatory signals acting through glucocorticoid receptor

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Reactive oxygen species-mediated activation of ASK1-p38 MAPK pathways is required for proinflammatory responses to the mycobacterial PPD antigen.

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Macrophage signaling upon mycobacterial infection: the role of mitogen-activated protein kinase pathways

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Protein kinase C zeta plays an essential role for Mycobacterium tuberculosis-induced extracellular signal-regulated kinase 1/2 activation in monocyte