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Role of peroxiredoxin II in the regulation of proinflammatory responses to LPS and the protection of mice from endotoxin-induced lethal shock

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The ginsenoside metabolite compound K inhibits toll-like receptor(TLR)-4 and TLR9-mediated inflammatory signals acting through glucocorticoid receptor

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Intracellular network of phosphatidylinositol 3-kinase, mammalian target of the rapamycin/70-kDa ribosomal S6 kinase 1, and mitogen-activated protein

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Protein kinase C zeta plays an essential role in mycobacteria-induced ERK 1/2 activation and expression of TNF-α and CC chemokines in human monocytes/

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Enhanced sensitivity to endotoxemia in the absence of peroxiredoxin II through NADPH oxidase-dependent activation of the MAPK and NF-κB pathways via T