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A progression of breast cancer appears to be inhibited by the treatment with a putative estrogenic agent, triclosan, via upregulation of cyclin D and cathepsin in female nude mice

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An interaction of estrogen receptor and insulin-like growth factor 1 receptor signaling pathways is involved in the anticancer effect of genistein on ovarian cancer growth induced by 17 β-estradiol or bisphenol A via the suppression of the crosstalk

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Chemo- and immune-therapies using engineered stem cells via upregulation of type I interferon alpha/beta receptor and BAX genes in a ductal breast cancer mouse model

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Treatment with octylphenol can lead to stimulate breast cancer cell growth via the alterations of cyclin D1 and p21 genes in human breast cancer cells through an estrogen receptor-mediated signaling pathway in breast cancer xenografted mice

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Transforming growth factor-beta signaling pathway was suppressed in the tumor progression caused by 4-nonylphenol and bisphenol A in BG-1 ovarian cancer xenografted mice