,

Triclosan, a potential estrogenic chemical, induced breast cancer progression expressing estrogen receptor via upregulation of cyclin D and cathepsin in a xenograft mouse model

,

A cross-talk between estrogen receptor alpha and insulin？ike growth factor receptor was inhibited by resveratrol on the growth of ovarian cancer stimulated by bisphenol A

,

A progression of primary and metastatic breast cancers was suppressed by engineered stem cells expressing therapeutic genes in a xenograft mouse model

,

Disruption of cell cycle regulation was caused by octylphenol in breast cancer cells via an estrogen receptor-dependent pathway in a cellular and xenograft mouse model

,

Tumor growth was suppressed by therapeutic stem cells expressing suicide gene and interferon-beta in endometrial cancer bearing SCID mice