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A potential endocrine disrupting chemical, 4-nonylphenol, stimulated the ovarian cancer cell growth by up-regulating cell cycle via an estrogen receptor signaling pathway in cellular and animal models

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Antitumor effect of endometrial cancer mass was induced by therapeutic stem cells fused cytosine deaminase and interferon-beta in a nude mouse model

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Bisphenol A-induced cell growth of BG-1 ovarian cancer cells was reversed by resveratrol, a phytoestrogen, via a down regulating ERα and IGF-1R cross-talk

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Exposure of phthalate stimuated the growth of LNCaP prostate cancer cells via estrogen receptor or androgen receptor-dependent signlaing pathway in cellular and xenograft mouse models

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Bisphenol A may induce a risk of ovarian cancer progression via an estrogen receptor pathway in cellular and animal models