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Exposure of various endocrine disrupting chemicals resulted in the proliferation of prostate cancer cells by regulating transforming growth factor-beta signaling pathway

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Resveratrol surppresed the cell proliferation induced by 17β-estradiol and bisphenol A by downregulating the cross-talk of estrogen receptor α and insulin-like growth factor-1 receptor in BG-1 ovarian cancer cells

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Potential effect of bisphenol A, an endocrine disrupting chemical, on the progression of ovarian cancer cell growth in cellular and xenograft mouse models

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Effects of therapeutic stem cells expressing cytosine deaminase and interferon-beta or carboxyl esterase on the progression of LNCaP prostate cancer cells

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Potential Estrogenicity or Tumorigenicity of Endocrine Disrupting Chemicals and Their Underlying Mechanism(s) in Cellular and Animal Models